|
|
|
Click Here to return To Results
|
|
We describe a clinical syndrome of delayed dystonia in children subsequent to initial gastrointestinal symptoms and acute noninflammatory encephalopathy.The syndrome was caused by the ingestion of mildewed sugarcane containing the Arthrinium-produced mycotoxin 3-nitropropionic acid(3-NPA).In the severely affected patients,intoxication usually was heralded by coma,with dystonia appearing 7 to 40 days after recovery from the coma.The dystonia was manifested as choreoathetosis,torsion spasms,or painful paroxysmal spasms of the extremities and was neither progressive nor reversible.CTs of the dystonic patients consistently showed bilateral hypodensities in the lenticular nuclei.The pathogenesis of the selective lenticular lesions induced by 3-NPA is not yet clear. |
|
(click to filter results - removes previous filter)
CAT scan CAT scan,abnormal coma dystonia dystonia,delayed onset encephalopathy lenticular nucleus,lesion of lenticular nucleus,lesion of,bilateral movement disorder MRI MRI,abnormal mycotoxin neurotoxin nitroproprionic acid seizure striatum,lesion of sugarcane,mildewed toxic encephalopathy toxins,nervous system
|
Click Here to return To Results
|
|