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While presentations may vary in individual patients, the essentials of history, clinical examination, laboratory evaluation, and neuroimaging lesion pattern can be efficiently directed to differentiate the various etiologies of deep gray matter nuclei lesions. In this review we focus on the etiologic classification and diagnostic approach top acute and chronic conditions that manifest on neuroimaging with bilateral symmetric lesions of deep gray matter nuclei. |
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basal ganglia,lesion of basal ganglia,lesion,bilateral carbon monoxide poisoning CAT scan CAT scan,abnormal caudate nucleus,lesion of cerebral venous thrombosis,deep children cyanide poison deep gray nuclei Hallervorden Spatz disease human immunodeficiency virus type 1 lenticular nucleus,lesion of,bilateral MELAS syndrome methanol intoxication MRI MRI,abnormal MRI,CAT scan compared to MRI,diffusion weighted neurofibromatosis 1 neurologic disease,diagnoses of neurotoxin putamen,lesion of,bilateral review article striatal encephalitis striatum,lesion of,bilateral subependymal nodules symmetric brain lesions thalamus,lesion of-bilateral tuberous sclerosis uremic encephalopathy
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